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Meteorological Factors and Air Pollutants Contributing to Seasonal Variation of Acute Exacerbation of Atrial Fibrillation A Population-Based Study

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dc.contributor.authorAhn, Jinhee-
dc.contributor.authorUhm, Taewoong-
dc.contributor.authorHan, Junhee-
dc.contributor.authorWon, Kyung-Mi-
dc.contributor.authorChoe, Jeong Cheon-
dc.contributor.authorShin, Ji Yeon-
dc.contributor.authorPark, Jin Sup-
dc.contributor.authorLee, Hye Won-
dc.contributor.authorOh, Jun-Hyok-
dc.contributor.authorChoi, Jung Hyun-
dc.contributor.authorLee, Han-Cheol-
dc.contributor.authorCha, Kwang Soo-
dc.contributor.authorHong, Taek Jong-
dc.contributor.authorKim, Young-Hoon-
dc.date.available2020-11-02T06:50:28Z-
dc.date.issued2018-12-
dc.identifier.issn1076-2752-
dc.identifier.issn1536-5948-
dc.identifier.urihttps://scholarworks.korea.ac.kr/kumedicine/handle/2020.sw.kumedicine/2889-
dc.description.abstractObjective: We investigated seasonal variation of acute exacerbation of atrial fibrillation (AAF) and contributing environmental factors. Methods: AAF events, meteorological elements, and air pollutants in Seoul between 2013 and 2015 were obtained from the nationwide database. AAF was defined if a patient visited the emergency room due to any AF-relevant symptoms or signs. Results: AAF occurred less frequently in summer than in other seasons (6.71 vs 7.25 events/d, P = 0.005). AAF tended to decrease with an increase of air temperature (r = -0.058). Among air pollutants, NO2 was significantly lower in summer and positively correlated with AAF after adjusting for other variables (b = 3.197). Conclusions: The rate of AAF events was the lowest in summer; air temperature and NO2 were contributing factors. The weather and environmental conditions should be considered as risk factors of AAF.-
dc.format.extent5-
dc.language영어-
dc.language.isoENG-
dc.publisherLIPPINCOTT WILLIAMS & WILKINS-
dc.titleMeteorological Factors and Air Pollutants Contributing to Seasonal Variation of Acute Exacerbation of Atrial Fibrillation A Population-Based Study-
dc.typeArticle-
dc.publisher.location미국-
dc.identifier.doi10.1097/JOM.0000000000001449-
dc.identifier.scopusid2-s2.0-85058536469-
dc.identifier.wosid000455900800012-
dc.identifier.bibliographicCitationJOURNAL OF OCCUPATIONAL AND ENVIRONMENTAL MEDICINE, v.60, no.12, pp 1082 - 1086-
dc.citation.titleJOURNAL OF OCCUPATIONAL AND ENVIRONMENTAL MEDICINE-
dc.citation.volume60-
dc.citation.number12-
dc.citation.startPage1082-
dc.citation.endPage1086-
dc.type.docTypeArticle-
dc.description.isOpenAccessN-
dc.description.journalRegisteredClasssci-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.relation.journalResearchAreaPublic, Environmental & Occupational Health-
dc.relation.journalWebOfScienceCategoryPublic, Environmental & Occupational Health-
dc.subject.keywordPlusSYMPATHETIC-NERVOUS-SYSTEM-
dc.subject.keywordPlusMYOCARDIAL-INFARCTION-
dc.subject.keywordPlusRESPIRATORY-DISEASE-
dc.subject.keywordPlusHEART-DISEASE-
dc.subject.keywordPlusCOLD-EXPOSURE-
dc.subject.keywordPlusPOLLUTION-
dc.subject.keywordPlusTEMPERATURE-
dc.subject.keywordPlusINCREASES-
dc.subject.keywordPlusRISK-
dc.subject.keywordPlusMORTALITY-
dc.subject.keywordAuthorair pollution-
dc.subject.keywordAuthoratrial fibrillation-
dc.subject.keywordAuthorexacerbation-
dc.subject.keywordAuthormeteorology-
dc.subject.keywordAuthortemperature-
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